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denosumab ke yong yu yu fang jue jing hou fu nv gu zhi shu song suo zhi gu zhe
Author(s): 
Pages: 2735
Year: Issue:  38
Journal: National Medical Journal of China

Abstract:  骨质疏松症的主要发病机制是机体的骨重建失衡,骨重建是指去除骨骼局部旧骨代之以形成新骨,即破骨细胞与成骨细胞一个成对的、相耦联的细胞活动过程,骨重建是成熟骨组织的一种重要替换机制.细胞核因子kB配体活化因子受体(RANKL)对破骨细胞的生长发育及功能有着极其重要的作用.
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